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5 Javita Videos You Must Share

Sharing tools and knowing which tools to share at the right time makes all the difference when securing a prospect. Tools help tell the story – the same story over and over again – about Javita and its products. Javita’s videos are an engaging and interactive part of your share process to introduce Javita to others.

Javita videos, like brochures, personal websites and magazines, are just as valuable as some of our other tools and are a great way to kick off the introduction of our products and business.

 

Here are the top 5 videos you should be exposing to your prospects:

Dollar Coffee Club (animated) video:

The illustrated “Billy the goat” Dollar Coffee Club video is a great first exposure of the company
for anyone you may come across.

The Dollar Coffee Club video introduces someone new to what the Dollar Coffee Club is. No longer
should coffee lovers over pay for their morning cup,  instead by choosing Javita coffee – they can
enjoy healthy premium coffee for only $1 a cup!

 

 

Burn + Control video:

Burn + Control coffee is Javita’s #1 product!

Members and Customers alike love the taste, but most importantly love the results they’re experiencing by drinking it.

The Burn + Control video goes in depth about this product, highlighting the science – ingredients and herbs
– and the effects each have on the body. This impactful 3-minute video also showcases testimonials from people
who are experiencing real results by incorporating Burn + Control coffee into their everyday routine.

 

 

 

Opportunity Sizzle video:

Don’t let the opportunity pass you by!

The Opportunity Sizzle video is great for someone who is looking for a new and rewarding opportunity,
whether it be income, time freedom or incentives & rewards!

This 1:30 minute video highlights Javita Members as they talk about the success they are experiencing
just by owning their own DCC business.

 

 

 

Product Sizzle video:

Let your prospects know that it’s time to, ‘Rethink Your Drink’!

With more than 150 million sticks (and counting) sold worldwide, Javita’s one-of-a-kind formulas are changing lives every day!

The Product Sizzle video is quick, fast-hitting, informative and factual. This video explores the amazing benefits Javita’s products offer.

 

 

 

Before & After Results:

The Before & After Results video showcases the before and after pictures of people who are using Javita products.
This video is so impactful the pictures speak for themselves.

 

 

 

Bonus video:

Now that you have secured your prospects to attend your party, here’s the perfect video to play.

What is the Dollar Coffee Club?

The “What is the Dollar Coffee Club?” video, which features Javita’s CEO Stan Cherelstein, is the perfect video to play
when hosting your next party. This video gives prospects the information they need to know about the company,
the power of owning your very own Dollar Coffee Club and what it can do for your life.

 

 

These videos can be viewed and shared by going to Javita.com/share or on the Javita App (available on iOS and Android).

Remember, every successful business is built by using the power of tools.

Enjoy!


Beta glucan induces proliferation and activation of monocytes in patients with advanced breast cancer

ABSTRACT

Glucans are glucose polymers that constitute a structural part of fungal cell wall. They can stimulate the innate immunity by activation of monocytes/macrophages. In human studies it has been shown that beta glucan has an immunomodulatory effect and can increase the efficacy of the biological therapies in cancer patients. In this prospective clinical trial we assessed in vivo effects of short term oral beta glucan administration on peripheral blood monocytes and their expression of activation markers in patients with advanced breast cancer.

Methods

23 female patients with advanced breast cancer were included in the study. Median age of the patients was 52 years. Sixteen healthy females with a median age of 48 years served as the control group for comparing the initial blood samples. Peripheral blood samples were drawn on day zero and patients started receiving oral 1–3, 1–6, D-beta glucan daily. Blood samples were recollected on the 15th day. In the initial samples mean lymphocyte count was significantly lower in the patients with breast cancer (1281 ± 306/mm3 versus 1930 ± 573/mm3, p = 0.04). In the patients with breast cancer, mean monocyte count which was 326 + 124/mm3 at the beginning, was increased to 496 + 194/mm3 at the 15th day (p = 0.015). Expression of CD95 (Apo1/Fas) on CD14 positive monocytes was 48.17% at the beginning, which was increased to 69.23 % at the 15th day (p = 0.002). Expression of CD45RA on CD14 positive monocytes was 49.9% at the beginning; it was increased significantly to 61.52% on day 15 (p = 0.001).

Conclusion

Oral beta glucan administration seems to stimulate proliferation and activation of peripheral blood monocytes in vivo in patients with advanced breast cancer.

Click here to access the original study.

 


Discover ‘Must Have’ Javita Products (Audio Recording)

Tune in as Javita’s Director of Botanical Research & Development Monica Johnson explores the advantage of using natural botanicals to address cardiovascular health, immune protection and joint mobility. All of the science and desired results without the side effects or steep price tag.


Phytosterol-enriched milk lowers LDL-cholesterol levels in Brazilian children and adolescents

 

Abstract

BACKGROUND AND AIMS:

Despite evidence of the lipid-lowering effect of plant sterols among adults with hypercholesterolemia, data regarding phytosterol use in children are limited. In this paper, we examined the effects of daily consumption of a phytosterol-enriched milk compound on the lipid profiles of Brazilian children and adolescents with dyslipidemia.

METHODS AND RESULTS:

This was a randomized, double blind, crossover clinical trial. Twenty eight dyslipidemics outpatients (aged 6-9 years) from an University Hospital were randomly allocated to control or intervention group. The intervention group received milk enriched with 1.2 g/day of plant sterol and the control group received the equivalent amount of skim milk during the period of 8 weeks. Changes from baseline in the mean lipid profile, including total cholesterol (TC), low density lipoprotein cholesterol (LDL-C), and triglyceride (TG) concentrations. Serum lipid profiles, glucose levels, dietary and anthropometric data were determined at weeks 0, 4, 8, 16, and 20.

Details regarding the safety and tolerance of phytosterol were obtained, using an open-ended questionnaire. Intention-to-treat analysis were performed, using the proc mixed procedure in SAS. After 8 weeks, the mean concentrations of TC and LDL-C were significantly reduced in the intervention group as compared to the control group with reductions of 5.9% (p = 0.09) and 10.2% (p = 0.002), respectively. In addition, TG concentrations were reduced by 19.7% (p = 0.09). No serious side effects were reported during the study.

CONCLUSION:

Our results confirm that plant sterols are an effective and safe treatment of infantile dyslipidemia.

Here is a link to the original study.


Protect Your Joints from AutoImmune Attack

More than 52 million Americans suffer from some form of arthritis.

Conventional medical wisdom has long held that rheumatoid arthritis results from an autoimmune attack on joints, while osteoarthritis was thought to result from age-related “wear-and-tear.”

For the first time, a team of researchers at Stanford University has demonstrated that this is not true!

It turns out that osteoarthritis is accompanied by the same pathological, pro-inflammatory immune factors involved in rheumatoid arthritis. Even more compelling was their finding that, if treatment is initiated before symptoms manifest, osteoarthritis may be entirely prevented.

Unfortunately, the list of available drugs to combat autoimmune disorders—including long-term treatment with corticosteroids like prednisone—is notoriously limited and comes with life-threatening complications, including obesity and diabetes.

The exciting news is a novel intervention has been identified that safely regulates the immune system to protect aging joint tissue from autoimmune attacks.

In this article, you will learn about natural collagen concentrate (NCC). Its unique molecular characteristics prevent immune cells’ overreaction to proteins normally found in cartilage and joint tissue that lead to pain and stiffness in both rheumatoid and osteoarthritis.

In multiple clinical trials using this proprietary collagen formulation, scientists at Harvard have been able to achieve relief of arthritic symptoms, with some patients experiencing complete remission!

You will also learn how NCC’s mode of action may be synergistically enhanced when combined with Boswellia serrata and two other joint-renewing nutrients.

The Stanford team’s discovery of the autoimmune link between osteoarthritis and rheumatoid arthritis was first presented in late 2011.

A group of 25 scientists concluded that the development of osteoarthritis is in great part driven by low-grade inflammatory processes. Specifically, the researchers discovered that the body launches an orchestrated, powerful attack on the synovial joints via signaling proteins normally used to fight infections. This autoimmune response, they reported, plays a key role in osteoarthritis onset.

Synovial joints are the most common joint types in the human body. They contain soft-tissue cushioning in addition to cartilage, along with synovial fluid, a natural lubricant. Knees, hips, and shoulders are just a few of the commonly arthritic joints that fall into this category.

What the Stanford team found was that low-grade inflammation is not merely an early symptom of osteoarthritic cartilage destruction in synovial joints—it is the trigger that causes it. The study also revealed that by targeting the autoimmune derangements that occur early on in the development of osteoarthritis, arthritis might be completely preventable.

They went on to point out that drugs intended to inhibit the arthritic reaction (like corticosteroids) paradoxically compromise the immune system. It would be far safer, they reported, if a natural way to turn off the body’s abnormal response were available.

Novel Method to Target the Pathologic Immune Response

Here’s how it works.These compelling new findings coincided with the development of a naturalintervention shown to protect tissues in aging joints called undenatured type II collagen.

Joints are lined with cartilage that both lubricates joints and absorbs physical impacts, preserving ease of motion and comfort. The bulk of the cartilage in your joints consists of collagen, a biomolecular protein critical to reducing friction and keeping joints youthful.

Osteoarthritis and rheumatoid arthritis both involve the structural degradation and gradual destruction of cartilage in aging joints. Osteoarthritis was long thought to be a consequence of simple “wear- and-tear” on joints (and hence largely inevitable). Rheumatoid arthritis, on the other hand, was recognized as an inflammatory autoimmune disease that arises when the body mistakenly attacks its own tissues, in this case the joint linings and cartilage.

We now know that both arise from pro-inflammatory immune factors.

Intrinsic to this process is the mobilization of “killer” T-cells into the joints by exposed collagen within the synovial lining.

Under normal conditions, collagen elicits no immune response. It is exposed collagen that immune cells mistakenly identify as invasive, foreign molecules.7 In response to the “threat,” inflammatory cytokines are released that draw in more “killer” T-cells.13 Those cells bombard exposed cartilage with toxic chemicals in order to destroy it, creating oxidative stress and further inflammation in the process.

Over time, these continuous biomolecular insults erode and disintegrate the cartilage that lubricates and functions as a shock-absorber in joints.

The resulting pain can become chronic and debilitating in lockstep with sensations of friction or grinding involved in joint movement. While less acute at rest, this pain is exacerbated by walking, standing, or any form of weight-bearing. Osteoarthritis sufferers often experience joint stiffness or immobility after periods of inactivity, for instance upon waking or after a long period of sitting.

NCC® Triggers Specific Oral Tolerance for Lasting Joint Relief

In both osteoarthritis and rheumatoid arthritis, the chief cause of autoimmune response is exposed collagen and the ensuing attack by sensitized killer T-cells.

Suppose an effective means of regulating killer T-cells before they encountered exposed collagen could be developed? This would “re-train” them to treat exposed collagen as a harmless substance and prevent joint degradation and destruction.

In 2000, the first hint of just such an intervention emerged.

A team of scientists at the University of Nebraska was surprised to find that chicken soup prevented the mobilization of immune system cells to sites of inflammation. Upon further analysis, it was not vegetables but a soluble component of the chicken broth itself that exerted this anti-inflammatory activity.

The researchers believe that it was likely the collagen from chicken bones in the broth that was the source of this beneficial anti-inflammatory effect.

New Way to Combat Rheumatoid and Arthritis

  • The most common joint disease, osteoarthritis, was long viewed as a disease of wear-and-tear. New findings reveal that it is one of a number of disorders caused by an abnormal response of the immune system.
  • Instead of the traditionally prescribed NSAIDs and immune suppressants, a revolutionary and side effect-free form of undenatured type II collagen called NCC® has been shown to desensitize the immune system and prevent pro-inflammatory autoimmune attacks on aging joints.
  • Undenatured type II chicken collagen has been shown in studies to be capable of activating the pathway known as induced oral tolerance, which teaches the immune system to correctly recognize exposed cartilage proteins as the body’s own tissues—instead of foreign microbes—thus preventing an inflammatory attack, a newly recognized cause of osteoarthritis.
  • The anti-inflammatory action of a novel composition of AKBA-enriched Boswellia serrata—and two joint-protective nutrients, glucosamine sulfate and boron, now available in highly bioactive formulations—can further boost the ability of undenatured type II chicken collagen to fight osteoarthritis, the painful condition behind so many visits to primary care physicians. Owing to its particular molecular structure, the chicken-derived type II collagen in UC-II® acts as a kind of “reverse vaccine,” one that regulates the immune system so that it stops mobilizing attacks against proteins normally found in healthy joint cartilage.

It does so by inducing what immunologists call specific oral tolerance—the desensitization of immune response to specific agents via an orally administered intervention. This is why NCC® may be likened to a kind of oral vaccine that reverses T-cell attacks on exposed cartilage.

Scientists at Harvard first studied the effects of NCC® on human patients with rheumatoid arthritis, long established as an autoimmune disorder. In a randomized, double-blind trial of 60 patients, undenatured type II chicken collagen produced a significant decrease in the number of swollen and painful joints within 3 months, compared to the placebo group. In fact, 14% of patients achieved complete remission. No side effects were found.

Later, a much larger study of 274 rheumatoid arthritis patients confirmed this finding, as did a study on patients with juvenile rheumatoid arthritis, a particularly aggressive form of this disease.

Turning their attention to osteoarthritis, scientists tested undenatured type II collagen on horses and dogs. They noted a reduction of 88% in measurable pain among horses given this formulation. In one study, moderately arthritic dogs given the undenatured collagen formulation were able to place more weight on sore limbs, and use them more naturally, relative to those given placebo, or those given chondroitin plus glucosamine.

The gold standard of scientific evidence is a randomized, double-blind, clinical study on humans. In a study of this type involving 52 adult human volunteers with osteoarthritis, who had an average age of 59, scientists found that in just 90 days, undenatured type II chicken collagen produced “significant enhancement in daily activities suggesting an improvement in their quality of life.”

In this trial, using the standardized WOMAC (Western Ontario McMaster Osteoarthritis Index) scale, scientists found that 40 mg a day of undenatured type II chicken collagen reduced osteoarthritis symptoms by 33% in 90 days. By comparison, the combination of 1,500 mg a day of glucosamine and 1,200 mg a day of chondroitin sulfate reduced WOMAC scores by only 14%.

Pain scores on the visual analog scale (VAS) decreased 40% for the collagen group, while pain scores for the glucosamine/chondroitin group decreased just 15%.

Finally, using the Lequesne’s functional index score—which measures pain during daily activities, such as walking—the study team found that undenatured type II collagen reduced this score by 20%, while the combination of glucosamine and chondroitin lowered the score by only 6%. All results were observed in just 90 days.

Why Undenatured Type II Collagen

As discussed earlier, immune system T-cells are tasked with recognizing and distinguishing between “self” molecules and “foreign” ones. They do this important work by responding to very specific molecular shapes and 3-dimensional structures. If T-cells in the blood are simply exposed, without any “training,” to a previously unrecognized protein structure (such as those found on joint collagen) they react violently and trigger a massive inflammatory response to destroy the protein.

This is why, when scientists want to create an animal model of arthritis, they inject collagen into their subjects, sensitizing the T-cells in their blood to the protein. Those circulating T-cells set up inflammation in the animal’s joints, with their rich supplies of collagen.

If T-cells are given adequate preparation, however, they can be “taught” that a specific molecule is a friend rather than a foe. Where does such T-cell “training” take place?

In the intestinal tract, specifically the lower end of the small intestine, which is rich in collections of immune tissue called Peyer’s patches. Peyer’s patches act as “training centers” for T-cells, exposing them to all sorts of molecular shapes that are natural components of the food we eat. In that fashion, we desensitize our immune systems and develop a natural tolerance to new foods without having constant allergic or inflammatory reactions.

So, by providing native collagen of the right 3-dimensional structure to the digestive tract, rather than to the bloodstream directly, we can “educate” our T-cells to ignore collagen when they encounter it in joints. Scientists say that this enables people to develop oral tolerance to collagen.

And oral tolerance to collagen powerfully suppresses joint inflammation, as has been shown in numerous laboratory studies. Oral administration of soluble type II collagen even prevents arthritis induced experimentally by collagen injections.

But not just any collagen works. Typical commercial processing causes collagen to become denatured, uncoiling from its normal helical shape and losing its 3-dimensional structure. Denatured collagen has no beneficial effects on joint inflammation.

A more natural form of collagen, called undenatured type II collagen, or UC-II®, has recently been developed. UC-II® is a highly effective product and a rich source of natural collagen. UC-II® retains its original 3-dimensional molecular structure, keeping it recognizable by T-cells in Peyer’s patches. And UC-II is robust enough to survive the harsh conditions in the stomach and small intestine, arriving at Peyer’s patches with its molecules intact.

Neutralizing the Pro-inflammatory 5-LOX Enzyme

Incorporating a safe anti-inflammatory agent in a joint protection program may provide an additional layer of defense against arthritic damage and pain, by helping to eliminate the immune trigger.

In traditional Indian medicine, the gum resin of Boswellia serrata is associated with alleviating inflammatory diseases such as arthritis. Double-blind, placebo-controlled studies have shown boswellia decreases swelling and pain in patients with osteoarthritis of the knee.

Various compounds within boswellia exert an anti-inflammatory action that is different from most anti-inflammatory agents: they inhibit the pro-inflammatory enzyme 5-lipoxygenase or 5-LOX.

A highly bioactive boswellia compound—called 3-O-acetyl-11-keto-b-boswellic acid, or AKBA—directly binds to and selectively inhibits 5-LOX.21,22 This prevents 5-LOX from facilitating the production of leukotriene, a pro-inflammatory compound that damages cartilage and joints. AKBA also reduces pro-inflammatory leukocyte elastase activity. The problem up to now has been limited bioavailability of AKBA following oral administration.

Fortunately, researchers have developed a boswellia formulation with enhanced bioavailability. Scientists administering this patented boswellia compound to animals found that it increased the bioavailability of AKBA in the systemic circulation by 52%, compared with a standard boswellia extract.

The researchers concluded that the AKBA-rich boswellia product “consistently…confers better anti-inflammatory efficacy,” and “provides more potential benefits in recovering articular cartilage damage… due to inflammatory insult in arthritis such as osteoarthritis or rheumatoid arthritis.”

In a double-blind, randomized, placebo-controlled study on human patients with osteoarthritis, 100 mg of the patented AKBA-enriched boswellia extract inhibited the cartilage-degrading enzyme MMP-3, and exhibited an anti-inflammatory action that was superior to a standard boswellia extract. Benefits were seen as early in the 90-day study as 7 days. The journal-published report described the formulation as a “novel synergistic composition.”


Bacopa Monnieri Offers Protection Against Alzheimer’s Disease

Alzheimer’s disease (AD) is a progressive neurodegenerative disease of the elderly. The rapid increase in its incidence has necessitated development of newer drugs. Ayurvedic herbal medications are increasingly researched due to their biosafety profile and usefulness in cognitive impairment. In this article, we critically reviewed one such Medhya Rasayana (nootropic drug) Brahmi-derived from extract of Bacopa monnieri (EBm).

Studies have shown that EBm promotes free radical scavenger mechanisms and protects cells in prefrontal cortex, hippocampus, and striatum against cytotoxicity and DNA damage implicated in AD. It also reduces lipoxygenase activity reducing lipid peroxidation, increases glutathione peroxidase and chelates iron. Administration of EBm was seen to protect the cholinergic neurons and reduce anticholinesterase activity comparable to donepezil, rivastigmine, and galantamine. It also reduces hippocampal β-amyloid deposition and stress-induced hippocampal damage. The neuroprotective effect of EBm is also due to nitric oxide-mediated cerebral vasodilation. EBm improved the total memory score and maximum improvement was seen in logical memory and paired associate learning in humans and reversed phenytoin-induced memory impairment in experimental model. EBm has not shown any serious clinical, neurological, hematological complications, or vital organs damage in experimental studies.

Amnesia and cognitive defects are cardinal symptoms of Alzheimer’s Disease (AD) []. In many studies conducted on humans and animals, Brahmi has shown to improve memory performance and cognitive function. In a double-blind, randomized, placebo-controlled trial conducted in Lucknow in India, 35 subjects aged above 55 years were subjected to Wechsler Memory Scale for comparison between placebo and EBm treatment groups. Subjects were tested on various sub-tests like general information, orientation, mental control, logical memory, digit forward, digit backward, visual reproduction, and paired associated learning. Scores were given to each sub-test and total memory score was calculated by adding the score of all subtests. The test results showed that there was significant improvement in total memory score of EBm-treated patients vs. placebo-treated patients. At 8 and 12 weeks after initiation of trial, maximum increment was seen after 8th week with maximum improvement seen in logical memory and paired associate learning sub-tests []. This study suggests that EBm can be useful agent in treatment of age-associated memory impairment.

In another study double-blind, randomized, placebo-controlled study efficacy of Brahmi as a memory enhancer on chronic dosing in 76 adults who were given Brahmi 300 mg/day and placebo. Subjects were tested for tasks of attention, memory, and psychological state at baseline. The results observed at 6 and 12 weeks after the trial demonstrated an increase in information retaining capacity over time. This was due to decreased forgetfulness as opposed to increased procurement because Brahmi did not show any beneficial effect on learning trials []. In a randomized, double-blind placebo-controlled trial of 36 children with Attention Deficit Hyperactivity Disorder, improvement in logical memory was demonstrated []. In a randomized, double-blind, placebo-controlled trial conducted in Australia on 81 subjects aged above 55 years who were given EBm in the dose of 300 mg/day for 12 weeks, remarkable improvement was demonstrated in verbal learning, memory acquisition, and delayed recall [].

Kumar et al. [] investigated the effect of EBm on cold stress induced neurodegeneration in hippocampus of rats. Histologically, rat brains were divided into 4 groups: group 1 consisted of rats which were kept in ideal laboratory conditions, group 2 rats were given EBm in the dose 40 mg/kg, group 3 rats were forced to swim in the cold water (temperature: 18 ± 2°C) for 1 month which generated cold water swim stress in their body, and group 4 were given cold water swim stress for 1 month which was followed by treatment by EBm for about 1 month in the dose of 40 mg/kg. Histophotometric study of hippocampus was done in which diameter of cells, total number of cells in the square, and packing density of cells were taken into consideration. Group 3 cells showed decreased diameter of cells, number of cells per square, and packing density of cells which was indicative of stress-induced damage while group 4 cells showed increased cell diameter, number of cells per square, and cell packing density. Group 4 rats showed the above parameters comparable to that of group 1 rats. This study demonstrated that EBm has got important therapeutic effect in abolishing stress-induced hippocampal damage.

In an experimental model by Saini et al. [], an intracerebroventricular injection of the drug colchicine was given to cause oxidative stress and increased lipid peroxidation, which resulted in significant memory loss. This was demonstrated by significant reduction in retention in elevated plus maze test. However, on treatment with EBm colchicine, administered animals showed a significant increase in retention time suggestive of cognitive improvement.

Antiepileptic drug phenytoin causes cognitive impairment on regular use in many patients. Using this principle, phenytoin was given to experimental rats in a dose of 25 mg/kg for 7 days resulting in significant cognitive impairment in the rats. Administration of EBm caused significant reversal of phenytoin-induced memory impairment [].

Benzodiazepines are known to cause dementia by their GABAergic action and by interfering with long-term potentiation. Diazepam was administered in a dose of 1.75 mg/kg to induce amnesia studied using the Morris Water Maze Test in mice. This was reversed by EBm given orally in a dose of 120 mg/kg [].

In a study conducted by Kishore and Singh [], it was found that Brahmi attenuated scopolamine, sodium nitrite and BN52021 (platelet activating factor antagonist) induced amnesia. The possible mechanism was by improving acetylcholine levels in mice in the above setting and was observed by improved performance on the Morris Water Maze Test. In this study, bacoside treatment decreased escape latency time which indicates that bacosides have predominant action on attenuating anterograde amnesia.

#increasedattention #betterrecall #healthycoffee #dollarcoffeeclub


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